01Analytical cohort
A high prevalence of excess weight
28 of 32 observations (87.5%) were overweight or obese. Principal inflammatory measurements were available for 29 encounters.
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Anthropometric Adiposity and Systemic Inflammation
Article companion for “Anthropometric Adiposity and Systemic Inflammatory Markers and Response in Neurological Disease: An Exploratory Study of Cerebrovascular Ischaemic Events Associated to Carotid Atheromatous Processes”.
Adiposity × NLR association
Reported associationNo cross-domain association survived FDR correction.
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Article-based scientific overview
The study examined 32 neurological monitoring encounters. Adiposity and systemic inflammatory markers showed strong relationships within their own domains, but weak cross-domain associations.
02Cross-domain result
Twenty tests, no FDR-supported association
The smallest adjusted p-value was 0.626. Waist circumference and NLR showed an inverse coefficient (ρ = −0.350; p = 0.063).
03Clinical context
Complementary biological dimensions
NLR did not clearly distinguish CIE from other neurological disease (p = 0.247). The findings are hypothesis-generating.
ResultReported estimateArticle-supported statement
BMI × NLRρ = −0.285; p = 0.135; FDR = 0.646.Inverse association, not statistically supported.
Waist × NLRρ = −0.350; p = 0.063; FDR = 0.626.No evidence of a consistent positive gradient.
Within domainsBMI–WHtR: ρ = 0.860; leukocytes–neutrophils: ρ = 0.901.Strong internal relationships within each domain.
CIE comparisonMedian NLR 2.93 vs 3.93; p = 0.247; Cliff’s δ = −0.287.Substantial overlap between diagnostic groups.
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